# BME100 s2014:W Group3 L2

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# OUR TEAM

 Name: Lorelei Fyfe Name: Ana Kenia Viera Name: Adnan Alsharif Name: Ben Lenschow Name: Joseph Roskop

# LAB 2 WRITE-UP

## Descriptive Statistics

Experiment 1

 LPS Dosage (mg) Mean Inflammotin (pg/ml) Mean Age (Years) Standard Deviation Count Stardard Error 0 3.834 66.7 1.52301 10 .48162 5 8.932 68.1 1.59393 10 .50405 10 61.622 68 30.11069 10 9.52183 15 657.941 67.9 212.94298 10 67.33848

Experiment 2

 LPS Dosage (mg) Mean Inflammotin (pg/ml) Standard Deviation Count Stardard Error 0 10.516 2.22555 5 .99530 5 11.112 7.40288 5 3.31067

## Results

Experiment 1

We performed an ANOVA test and got a p-value of 1.401 * 10^-16. Since it was an ANOVA test we performed a Bonferroni Correction on our alpha value to get a new alpha value of .00833. This was statistically significant since our p-value is lower than our alpha value.

Experiment 2
A T-Test was used because there was one independent variable that only had two values. It was a comparison of two factors. The p-value found on 0mg versus 10mg was 0.867. With alpha being 0.05, this test was not statistically significant.

## Analysis

Experiment 1

Also a t-test between each comparison produced values ranging from .00003 to .000000018 which are all less than .00833 (our alpha level) so all comparisons are statistically significant. With an increase in dosage brought an increase in inflammotin levels which were all statistically proven correct.

Experiment 2
The difference in inflammotin levels for the rats given 0mg versus 10mg was not statistically significant. The difference in mg did cause a slight increase in the inflammotin levels of the rats with 10mg, but not enough to conclude that the dosage was the factor causing it.

## Summary/Discussion

The T-Test performed with the rats was not statistically significant while the ANOVA Test with humans was. This could be due to the larger sample size with humans or the fact that the humans were tested with much higher levels of the product. In humans, there was a consistent increase in inflammotin levels corresponding with an increase in dosage.