The p-value for the human study was 1.4e^-16. On the other hand, the rat study produced p-values of .433702 for the first set of data, and a value of .867403 for the second set of data. To put these numbers in context, the human p-value was substantially lower than the ideal p-value of .05, which concludes the test was extremely accurate. For the rat test, the p-value was , in both cases, higher than the ideal value of .05, which shows that this portion of the experiment was inconclusive.

Summary/Discussion

Based on the information gathered in the study, the averages, standard deviation, standard error, counts, and p-values were calculated. For the human trial, a single ANOVA was used to compare four sets of data, and in the rat trail a t-Test was used to compared two sets of data. With the collected data, a graph displaying the average value of inflammotin at each dosage of LPS (0mg, 5mg, 10mg, 15mg), verses the standard deviation was created. The graph of the humans showed that at LPS levels of 0mg, 5mg, and 10mg, the level of inflammotin stayed consistently under 100 pg/ml. Once the dosage was increased to 15mg, the inflammotin was increased to 650pg/ml. In the rat study, the level of inflammotin stayed relatively constant in both groups (0mg, and 10mg). The human study produced better results due to the amount and accuracy of the data. On the contrary, the rat data was inconclusive due to the lack of different dosage trials and inconsistent data. With that being said there is a definite positive correlation between LPS and humans, but not the same for rats.