# BME100 s2016:Group10 W1030AM L2

BME 100 Spring 2016 | Home People Lab Write-Up 1 | Lab Write-Up 2 | Lab Write-Up 3 Lab Write-Up 4 | Lab Write-Up 5 | Lab Write-Up 6 Course Logistics For Instructors Photos Wiki Editing Help
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## OUR TEAM WE THE BEST## LAB 2 WRITE-UP## Descriptive Statistics
The standard deviation within the group of rats given 0mg dosage was 2.23 pg/mL. The 10mg dosage rats had a standard deviation of 7.403 pg/mL. The standard error for the 0mg dosage group of rats was .9952 pg/mL. The group of rats given the dosage of 10mg had a standard error of 3.31 pg/mL.
The mean Inflammotin protein levels differ based on varying doses. For the 0mg dosage, the average Inflammotin level is 3.834 pg/mL. For the 5mg dosage, the average Inflammotin level is 8.932 pg/mL. For the 10mg dosage, the average Inflammotin level is 61.622 pg/mL. For the 15mg dosage, the the average Inflammotin level is 657.941 pg/mL. The standard deviation also differs based on dosages. The 0mg dosage yielded a 1.523 pg/mL standard deviation, the 5mg dosage yielded a 1.594 pg/mL standard deviation, the 10mg dosage yielded a 61.622 pg/mL standard deviation, and finally, the 15mg dosage yielded a 212.943 pg/mL standard deviation. The standard error similarly differs based on dosages. The standard error for 0mg is 0.482 pg/mL, the standard error for 5mg is 0.504 pg/mL, the standard error for 10mg is 9.521 pg/mL, and the standard error for 15mg is 67.338 pg/mL. Overall, the averages, standard deviations, and standard errors, all increase as the dosages increase.
## Results
## Analysis
## Summary/DiscussionThe claim that LPS increases the levels of inflammotin in the human subjects is valid because the result was statistically significant. This cannot be said for the rat trials, because the rat study was not statistically significant. The rat group was statistically insignificant, because the change in inflammotin was too small to be directly attributed to the LPS. This resulted in a p value greater than 0.05. The possible reasons for the difference in statistical significance could be in the size of the trial, since the human study had a larger pool of samples. Another potential reason for this is LPS has a profound biological impact on humans, but not as much on rats. It is difficult to identify why LPS increased inflammotin levels in humans, but not statistically significant in rats. Differences in anatomy, physical structures, and chemical signals can all play a role. There is also the ability that the data obtained many have been inaccurate because of experimental errors. Based on the analysis however, it is safe to say that there needs to be more experiments and trials run, before a precise conclusion can be made. |