# BME100 s2015:Group1 9amL2

BME 100 Spring 2015 Home
People
Lab Write-Up 1 | Lab Write-Up 2 | Lab Write-Up 3
Lab Write-Up 4 | Lab Write-Up 5 | Lab Write-Up 6
Course Logistics For Instructors
Photos
Wiki Editing Help

# OUR TEAM

 Name: Shawn Striker File:Gracekim.jpgName: Grace Kim Name: Jonathan Moreno Name: Ryan Meyer Name: Your name

# LAB 2 WRITE-UP

## Descriptive Statistics

Experiment 1
The first set of data is part of the human study.
Inflammotin mean for the human studies is included.

Details of the study:
Forty people between the age of 60 to 82 were chosen to participate in the study.
A total of four groups were tested and given different dosages.
Group one received 0 mg of LPS. Group two received 5 mg of LPS.
Group one received 10 mg of LPS. Group one received 15 mg of LPS.

Experiment 2
The second set of data is part of the rat study.

Experiment 1

Experiment 2

## Analysis

Experiment 1
The ANOVA test for the four experimental groups in the human study.

The ANOVA test resulted in a P-value of 1.4E^-16. This number is extremely close to zero and is also less than 0.05, which shows that at least some of the numbers were significant. The t-test was used on each relationship between the four groups.

Every t-test of each relationship resulted in a value that was less than the corrected P-value. This shows there was a significance between each group.

Experiment 2
The t-test resulted in a value of 0.867403. When compared to the value of 0.05, it is shown that the rat experiment had no significance between the two experimental groups.

## Summary/Discussion

In experiment 1, based on the data collected, all of the groups were significant to each other. In experiment 2, based on the data collected, all of the groups were insignificant to each other. We divided by 6 because there were 6 comparisons total. The study cannot be used with rats because the data was insignificant. Ideal subjects for this study would be humans as the Anova test revealed that the data for humans was significant. There was a strong correlation between inflammotin and LPS doses that was not seen in the rat data. There could be multiple factors for why the rat data was insignificant such as health/condition/age of the rats.

Upon further analyzation of the data from the human and rat experiments it can be concluded that the two experiments are actually linked to each other even though the human experiment was significant and the rat experiment was insignificant. The way that the two experiments is that the test subjects in both the experiments had varying levels of inflammotin in both those that did not receive LPS and those that did receive a dosage of LPS.