BME100 f2013:W900 Group13 L2

From OpenWetWare
Jump to navigationJump to search
BME 100 Fall 2013 Home
Lab Write-Up 1 | Lab Write-Up 2 | Lab Write-Up 3
Lab Write-Up 4 | Lab Write-Up 5 | Lab Write-Up 6
Course Logistics For Instructors
Wiki Editing Help


Name: Allison Marley
Name: Tyler Angell
Name: Cory Riecken
Name: Andrew Luc
Name: Reem Gerais
Name: student


Descriptive Statistics

Human Study Data

Rat Study Data


Experiment 1[Human Study]

Experiment 2[Rat Study]


Experiment 1 [Human Study]

Results from the experiment yielded a P-value of 1.40x10^-6 which is significantly lower than 0.05. From this, we took a post-hoc test to validate the significance between the individual dosages. Our data showed that there are significant differences between each of the dosages.

Experiment 2 [Rat Study]


Using statistics, we computed a P-value of approxiamtly .867. Therefore since our p-value is greater than .05, we have concluded that our p-value shows no significant difference between the two dosages in rats. (Using inferential statistics, please determine statistically significant differences in the data.)


Experiment 1 (Human Study): The analysis shows that there is a clear and present correlation between the dosage of lipopolysaccharide and inflammotin production in human subjects however there is a high variance among test subjects who took higher doses and so our data, analysis, and conclusion may be unreliable.

Experiment 2 (Rat Study): Our analysis shows no correlation between lipopolysaccharide dosage and inflammotin production in rats, but since there were so few test subjects, the results may not be representative of rats as a population and so more tests would need to be done with more subjects to find definite results.

From the results we can see that there is a large increase in inflammotin production in humans as the dosage of LPS increases, however we can see that the deviation between subjects increases in relation to the dosage. This large variance between subjects receiving higher doses means that more work needs to be done in order to have clear, reliable results. On the other hand there is little increase in inflammotin production in rats when the dosage of LPS increases, but like the humans there is an increase in the standard error as the dosage increases. This high deviation when there is a high dosage means that as the dosage increases, our data and consequently our results are less accurate. With that in mind, our analysis of the results in humans showed a p-value of 1.40E-16, which means that there is a definite difference in inflammotin production in relation to dosage of LPS, which means that increasing the dosage of LPS will have a significant impact on inflammotin production. In our analysis of the results in rats we found that the p-value= 0.8674035, which means there is little difference in inflammotin production between the different dosages of LPS, so increasing the dose of LPS will not cause a meaningful change in inflammotin production in rats.