BME100 f2013:W900 Group10 L2

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BME 100 Fall 2013 Home
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Lab Write-Up 1 | Lab Write-Up 2 | Lab Write-Up 3
Lab Write-Up 4 | Lab Write-Up 5 | Lab Write-Up 6
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OUR TEAM

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Name: Barrett Anderies
Role(s): Summary/Discussion
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Name: Joslin Jose
Role(s): Analysis
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Name: Takerra Waters
Role(s): Results
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Name: Duran Charles
Role(s)Discriptive Statistic
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Name: Liam Williams
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Name: student
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LAB 2 WRITE-UP

Descriptive Statistics

Experiment 1

TABLE 1: Results of data taken from human subjects representing the different inflammontin protein levels at four different dosage of lipopolysaccharide (LPS).

TABLE 2: The mean and standard deviation of the inflammontin protein levels at dosage levels (0mg, 5mg, 10mg, and 15mg) of LPS. Column graph of the data is plotted below in Results.



Experiment 2

TABLE 1: Results of data taken from rat study representing inflammontin protein levels the dosage of 0mg and 10mg of lipopolysaccharide

TABLE 2: The mean and standard deviation of the inflammontin protein level from dosage levels of 0mg and 10mg of lipopolysaccharide. Column graph of data is plotted below in Results.





Results

Experiment 1

The data on this graph shows the dosage of LPS given to Humans 60years old and older and the effect it has on their inflammation

Experiment 2

The data on this graph shows the dosage of LPS given to Rats and the effect it has on their inflammation




Analysis

Experiment 1

ANOVA:

Post-Hoc test


Experiment 2

T-Test:




Summary/Discussion

Human Study:

The analysis of the data shows a significant correlation between the dosage of lipopolysaccharide and the Inflammotin levels. The p-value from the ANOVA test shows that the largest within group variation is well within acceptable limits and the t-test shows a significant between group variation between all groups. Thus we can conclude that lipopolysaccharide does affect Inflammotin levels at all of the tested doses.

Rat Study:

The analysis of the data shows no correlation between the dosage of lipopolysaccharide and the Inflammotin levels. The t-test shows that there is no significance between group variation in the two dose groups. Therefore we have failed to disprove our null hypothesis and can say nothing about the correlation between lipopolysaccharide dose and Inflammotin levels in rats.