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[1] B. Jiang, F. Han, M.H. Lu, Z.P. Wang, W. Liu, Y.X. Zhang, J. Xu, and R.J. Li, Bis-chalcone polyphenols with potential preventive and therapeutic effects on PD: Design, synthesis and in vitro disaggregation activity against alpha-synuclein oligomers and fibrils. Eur J Med Chem, 2022. 239: p. 114529. http://doi.org/10.1016/j.ejmech.2022.114529

[2] F. Han, B. Jiang, M.H. Lu, Z.P. Wang, W. Liu, Y.X. Zhang, and J. Xu, Hybrids of polyphenolic acids and xanthone, the potential preventive and therapeutic effects on PD: Design, synthesis, in vitro anti-aggregation of alpha-synuclein, and disaggregation against the existed alpha-synuclein oligomer and fibril. Bioorg Med Chem, 2022. 66: p. 116818. http://doi.org/10.1016/j.bmc.2022.116818

[3] H. Zhang, C. Tan, X. Shi, and J. Xu, Impacts of autofluorescence on fluorescence based techniques to study microglia. BMC Neurosci, 2022. 23(1): p. 21. http://doi.org/10.1186/s12868-022-00703-1

[4] L. Chen, G.L. Huang, M.H. Lu, Y.X. Zhang, J. Xu, and S.P. Bai, Amide derivatives of Gallic acid: Design, synthesis and evaluation of inhibitory activities against in vitro alpha-synuclein aggregation. Bioorg Med Chem, 2020. 28(15): p. 115596. http://doi.org/10.1016/j.bmc.2020.115596

[5] H. Liu, L. Chen, F. Zhou, Y.X. Zhang, J. Xu, M. Xu, and S.P. Bai, Anti-oligomerization sheet molecules: Design, synthesis and evaluation of inhibitory activities against alpha-synuclein aggregation. Bioorg Med Chem, 2019. 27(14): p. 3089-3096. http://doi.org/10.1016/j.bmc.2019.05.032

Before ZZU:

[1] J. Xu, A.M. Bernstein, A. Wong, X.H. Lu, S. Khoja, X.W. Yang, D.L. Davies, P. Micevych, M.V. Sofroniew, and B.S. Khakh, P2X4 Receptor Reporter Mice: Sparse Brain Expression and Feeding-Related Presynaptic Facilitation in the Arcuate Nucleus. J Neurosci, 2016. 36(34): p. 8902-20.

[2] R. Srinivasan, T.Y. Lu, H. Chai, J. Xu, B.S. Huang, P. Golshani, G. Coppola, and B.S. Khakh, New Transgenic Mouse Lines for Selectively Targeting Astrocytes and Studying Calcium Signals in Astrocyte Processes In Situ and In Vivo. Neuron, 2016. 92(6): p. 1181-1195.

[3] J. Xu and B.S. Khakh, Slow neuromodulation mediated by ATP P2X receptors. Neuron, 2014. 83(2): p. 257-9.

[4] J. Xu, H. Chai, K. Ehinger, T.M. Egan, R. Srinivasan, M. Frick, and B.S. Khakh, Imaging P2X4 receptor subcellular distribution, trafficking, and regulation using P2X4-pHluorin. J Gen Physiol, 2014. 144(1): p. 81-104.

[5] X. Tong, Y. Ao, G.C. Faas, S.E. Nwaobi, J. Xu, M.D. Haustein, M.A. Anderson, I. Mody, M.L. Olsen, M.V. Sofroniew, and B.S. Khakh, Astrocyte Kir4.1 ion channel deficits contribute to neuronal dysfunction in Huntington's disease model mice. Nat Neurosci, 2014. 17(5): p. 694-703.

[6] M.D. Haustein, S. Kracun, X.H. Lu, T. Shih, O. Jackson-Weaver, X. Tong, J. Xu, X.W. Yang, T.J. O'Dell, J.S. Marvin, M.H. Ellisman, E.A. Bushong, L.L. Looger, and B.S. Khakh, Conditions and constraints for astrocyte calcium signaling in the hippocampal mossy fiber pathway. Neuron, 2014. 82(2): p. 413-29.

[7] J. Xu and B.J. Nicholson, The role of connexins in ear and skin physiology - functional insights from disease-associated mutations. Biochim Biophys Acta, 2013. 1828(1): p. 167-78.

[8] E. Shigetomi, E.A. Bushong, M.D. Haustein, X. Tong, O. Jackson-Weaver, S. Kracun, J. Xu, M.V. Sofroniew, M.H. Ellisman, and B.S. Khakh, Imaging calcium microdomains within entire astrocyte territories and endfeet with GCaMPs expressed using adeno-associated viruses. J Gen Physiol, 2013. 141(5): p. 633-47.

[9] Q. Ren, M.A. Riquelme, J. Xu, X. Yan, B.J. Nicholson, S. Gu, and J.X. Jiang, Cataract-causing mutation of human connexin 46 impairs gap junction, but increases hemichannel function and cell death. PLoS One, 2013. 8(9): p. e74732.

[10] S.S. Mitra, J. Xu, and B.J. Nicholson, Coregulation of multiple signaling mechanisms in pp60v-Src-induced closure of Cx43 gap junction channels. J Membr Biol, 2012. 245(8): p. 495-506.

[11] J. Liu, J. Xu, S. Gu, B.J. Nicholson, and J.X. Jiang, Aquaporin 0 enhances gap junction coupling via its cell adhesion function and interaction with connexin 50. J Cell Sci, 2011. 124(Pt 2): p. 198-206.

[12] I.L. Thibodeau, J. Xu, Q. Li, G. Liu, K. Lam, J.P. Veinot, D.H. Birnie, D.L. Jones, A.D. Krahn, R. Lemery, B.J. Nicholson, and M.H. Gollob, Paradigm of genetic mosaicism and lone atrial fibrillation: physiological characterization of a connexin 43-deletion mutant identified from atrial tissue. Circulation, 2010. 122(3): p. 236-44.

[13] C. Ambrosi, D. Boassa, J. Pranskevich, A. Smock, A. Oshima, J. Xu, B.J. Nicholson, and G.E. Sosinsky, Analysis of four connexin26 mutant gap junctions and hemichannels reveals variations in hexamer stability. Biophys J, 2010. 98(9): p. 1809-19.

[14] T.A. Starich, J. Xu, I.M. Skerrett, B.J. Nicholson, and J.E. Shaw, Interactions between innexins UNC-7 and UNC-9 mediate electrical synapse specificity in the Caenorhabditis elegans locomotory nervous system. Neural Dev, 2009. 4: p. 16.