# BME100 f2015:Group2 8amL2

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# LAB 2 WRITE-UP

## Descriptive Statistics

Experiment 1 (Human Study)
For the human study we were able to find the mean of the different dosages of inflammotin which go as follows: at 0 mg dosage the mean is 3.834 (pg/ml), at 5 mg dosage the mean is 8.932 (pg/ml), at 10 mg dosage the mean is 61.622 (pg/ml), at 15 mg dosage the mean is 657.941 (pg/ml). With this data we were also able to collect the standard deviation and standard error for this study. at 0 mg dosage the standard deviation was 1.52301018 and the standard error for this dosage is 0.48161811, at 5 mg dosage the standard deviation was 1.59393155 and the standard error for this dosage is 0.50404541, at 10 mg dosage the standard deviation was 30.1106939 and the standard error is 9.52183745, at 15 mg dosage the standard deviation was 212.942976 and the standard error is 67.3384817.

Experiment 2 (Rat Study)
For the rat study, the group was able to find the mean of each dosage of inflammotin: at 0 mg, the mean is 10.516 (pg/mL), at 10 mg, the mean is 11.112 (pg/mL).

With the previous means, the standard deviation and standard error were able to be calculated: at 0 mg, the standard deviation is 2.226 and the standard error is 4.703. at 10 mg, the standard deviation is 7.403 and the standard error is 3.311.

## Results

Experiment 1 (Human Study)

(Graph: Results of human Inflammotin study with standard deviation.)

Experiment 2 (Rat Study)

(Graph: Results of rat Inflammotin study with standard deviation.)

## Analysis

Experiment 1 (Human Study)

In the above image, the p-value can be obtained from the ANOVA test. The p-value is less than .05, therefore there is statistical difference between the groups.

Experiment 2 (Rat Study)

```    Since there are only 2 data sets for the rat study, the t-test was used.The p-value was found to be 0.8674035, which is greater than 0.05. Therefore,there is not any significance between the data sets.
```

## Summary/Discuss

From the study, you can see that the LPS has an effect on both the human subjects and the rat subjects. However, the inflammotin levels were different between the varied between the two groups. Even though the LPS did increase the amount of inflammotin in the rat subjects, the inflammotin increase was not very significant. In the human subjects, as the LPS increased, so did the inflammotin levels, and significantly too. Through our analysis of the information, we can conclude that LPS does increase inflammotin in humans. The more LPS present, the inflammotin is drastically increased. In rats, an increase in the amount of LPS only increases the amount of inflammotin very slightly.