Intracellular anandamide trafficking as a promoter of neurogenesis acting through presynaptic CB1/FABP binding
The maturation and differentiation of neural stem cells via fatty-acid binding proteins (FABPs) signaling may require anandamide as a promoter of CB1-dependent neurogenesis. 
Hippocampal neurogenesis with synaptogenic promoter (DHA) is induced by intracellular and presynaptic CB1 receptor binding to anandamide. 
- The allosteric activation of PPAR-RXR heterodimer by anandamide (CB1/TRPV1 receptors) may facilitate hippocampal neurogenesis. (Reference needed)
- Intracellular FABP5 expression may result in anandamide trafficking of heteromeric CB1 receptor. (Reference needed)
- Adenosine antagonism (caffeine) may potentiate retrograde trafficking of anandamide via heteromization of CB1 receptor. (Reference needed)
- Anandamide biosynthesis may enhance caffeine-induced diuresis. 
- Anandamide-oxytocin cross-talk (signal transduction) may modulate cellular stress response/vitamin D gene expression?
Type-1 (CB1) Cannabinoid Receptor Promotes Neuronal Differentiation and Maturation of Neural Stem Cells
Intracellular cannabinoid type 1 (CB1) receptors are activated by anandamide.
Diuretic Effects of Cannabinoids
The endocannabinoid anandamide inhibits cholangiocarcinoma growth via activation of the noncanonical Wnt signaling pathway.
The endogenous cannabinoid anandamide inhibits human breast cancer cell proliferation.