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| ===Neurotoxicity of coal fly ash nanoparticles===
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| * Combustion-derived nanoparticles (coal fly ash) are neurotoxic and may contain silicon dioxide, aluminium oxide, barium oxide, arsenic and dimethyl sulfate. <cite>Paper8</cite>
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| * [http://www.thenakedscientists.com/forum/index.php?topic=65238 Is geoengineering destroying life?]
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| * [http://www.thenakedscientists.com/forum/index.php?topic=49418 What are the consequences of being sprayed with barium and aluminium?]
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| * [http://www.thenakedscientists.com/forum/index.php?topic=67517 Are drug nanoparticles harmful in the same way that pollutants are?]
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| ===Synaptic hypercomputation===
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| * biological utilization of nonlocality and entanglement of water during exocytosis: Is quantum hypercomputation a function of synaptic exocytosis?
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| * [http://www.thenakedscientists.com/forum/index.php?topic=66293.0 Is quantum interference in brain activity possible?]
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| ====Phase coherence of synaptic hypercomputation====
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| * Retrograde signaling and synaptogenesis regulate phase coherence of synaptic exocytosis?
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| ===Retrograde signaling===
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| * <font color=red>Allosteric modulation of glycine receptors by endocannabinoids</font>
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| ** THC and anandamide may modulates synaptogenesis in the retina and brain through glycinergic transmission <cite>Paper2</cite>
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| ** DHA facilitate NMDA receptor activation in the presence of glycine <cite>Paper3</cite>
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| ** THC and anandamide enhance NMDA-dependent LTP through glycine receptor activation
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| ====CB1-induced cholinergic neurotransmission====
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| * Hypothalamic POMC neurons promote CB1-induced feeding <cite>Paper4</cite>
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| * Caffeine potentiate striatal neurotransmission through presynaptic CB1 receptor activation. <cite>Paper5</cite>
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| * Monoamine oxidase inhibition affects cannabinoid-induced synaptogenesis. <cite>Paper6</cite>
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| ====intracellular CB1/BDNF signaling====
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| * CB1 receptor activation induce striatal neuroprotection through a PI3K/Akt/mTORC1/BDNF pathway. <cite>Paper7</cite>
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| ===Political manipulations of psychiatry (Sluggish schizophrenia)===
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| * Modern psychiatry is a pseudoscience; Its goal is to alienate consciousness based on pseudoscientific voodoo.
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| ** Schizophrenia is a scam to subvert consciousness with antipsychotics (AP)
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| * '''"Psychiatry as a means of intimidation"''': http://schizophreniabulletin.oxfordjournals.org/content/36/1/33.full
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| ** Psychiatry is psychological oppression.
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| ===Ubiquity of consciousness===
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| * The brain is not the source of consciousness: http://www.tandfonline.com/doi/full/10.1080/19420889.2015.1085138
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| ** http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3245704/
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| * Intrinsic awareness (IA); A machine has no internal purpose within. http://arxiv.org/abs/1101.2422
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| ===References===
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| <biblio>
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| #Paper1 https://www.jstage.jst.go.jp/article/jts/36/3/36_3_267/_article
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| //Nasal instillation of nanoparticle-rich diesel exhaust particles slightly affects emotional behavior and learning capability in rats
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| #Paper2 pmid=16332990
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| //Delta9-tetrahydrocannabinol and endogenous cannabinoid anandamide directly potentiate the function of glycine receptors.
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| #Paper3 pmid=7514666
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| //Facilitatory effect of docosahexaenoic acid on N-methyl-D-aspartate response in pyramidal neurones of rat cerebral cortex.
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| #Paper4 pmid=25707796
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| //Hypothalamic POMC neurons promote cannabinoid-induced feeding.
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| #Paper5 pmid=19027757
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| //Caffeine drinking potentiates cannabinoid transmission in the striatum: interaction with stress effects.
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| #Paper6 pmid=23858446
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| //Cognitive abnormalities and hippocampal alterations in monoamine oxidase A and B knockout mice.
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| #Paper7 pmid=25698444
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| //The CB₁ cannabinoid receptor signals striatal neuroprotection via a PI3K/Akt/mTORC1/BDNF pathway.
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| #Paper8 pmid=7350652
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| //Dimethyl and monomethyl sulfate: presence in coal fly ash and airborne particulate matter.
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| #Paper9 http://particleandfibretoxicology.biomedcentral.com/articles/10.1186/1743-8977-2-10
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| //Combustion-derived nanoparticles: A review of their toxicology following inhalation exposure.
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| </biblio>
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