User:Andrew S. Gilder: Difference between revisions
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https://www.researchgate.net/profile/Andrew_Gilder/ | https://www.researchgate.net/profile/Andrew_Gilder/ | ||
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==Publications== | ==Publications== | ||
Gilder AS, Jones KA, Hu J, Wang L, Chen CC, Carter BS, Gonias SL. Soluble uPAR | |||
is released selectively by glioblastoma cells that express EGF receptor variant | |||
III and promotes tumor cell migration and invasion. J Biol Chem. 2015 Apr 2. pii: | |||
jbc.M115.637488. [Epub ahead of print] PubMed PMID: 25837250. [http://www.jbc.org/content/early/2015/04/02/jbc.M115.637488.long] | |||
Hu J, Jo M, Eastman BM, Gilder AS, Bui JD, Gonias SL. uPAR induces expression | |||
of transforming growth factor β and interleukin-4 in cancer cells to promote | |||
tumor-permissive conditioning of macrophages. Am J Pathol. 2014 | |||
Dec;184(12):3384-93. doi: 10.1016/j.ajpath.2014.08.003. Epub 2014 Oct 11. PubMed | |||
PMID: 25310970; PubMed Central PMCID: PMC4258500. [http://www.sciencedirect.com/science/article/pii/S000294401400488X] | |||
Coilin participates in the suppression of RNA polymerase I in response to cisplatin induced DNA damage. Mol Biol Cell. 2011 April 1; Vol. 22(7):1070-1079, PMID: 21289084 [http://www.molbiolcell.org/cgi/content/abstract/22/7/1070]Cover art: [http://www.molbiolcell.org/content/vol22/issue7/cover.shtml] | Coilin participates in the suppression of RNA polymerase I in response to cisplatin induced DNA damage. Mol Biol Cell. 2011 April 1; Vol. 22(7):1070-1079, PMID: 21289084 [http://www.molbiolcell.org/cgi/content/abstract/22/7/1070]Cover art: [http://www.molbiolcell.org/content/vol22/issue7/cover.shtml] | ||
Revision as of 11:00, 11 May 2015
Contact Info
- Andrew Gilder
- UC San Diego
- Department of Pathology
- 9500 Gilman Dr.
- La Jolla, CA 92093
- agilder@ucsd.edu
- Email me through OpenWetWare
https://www.researchgate.net/profile/Andrew_Gilder/
http://scholar.google.com/citations?user=MsyZ9MgAAAAJ
http://www.linkedin.com/pub/andrew-gilder/38/120/604
Currently a postdoctoral fellow at UC San Diego.
Hebert Lab Alumus (2011) [1].
Education
- 2007, B.S. Biology, Economics Millsaps College
- 2010, M.S. Biomedical Sciences, University of Mississippi Medical Center
- 2011, Ph.D. Biochemistry, University of Mississippi Medical Center
Research interests
- Control of gene transcription and splicing
- Nuclear body dynamics
- Epigenetics
- DNA damage response
- Cancer cell biology
Publications
Gilder AS, Jones KA, Hu J, Wang L, Chen CC, Carter BS, Gonias SL. Soluble uPAR
is released selectively by glioblastoma cells that express EGF receptor variant III and promotes tumor cell migration and invasion. J Biol Chem. 2015 Apr 2. pii: jbc.M115.637488. [Epub ahead of print] PubMed PMID: 25837250. [2]
Hu J, Jo M, Eastman BM, Gilder AS, Bui JD, Gonias SL. uPAR induces expression of transforming growth factor β and interleukin-4 in cancer cells to promote tumor-permissive conditioning of macrophages. Am J Pathol. 2014 Dec;184(12):3384-93. doi: 10.1016/j.ajpath.2014.08.003. Epub 2014 Oct 11. PubMed PMID: 25310970; PubMed Central PMCID: PMC4258500. [3]
Coilin participates in the suppression of RNA polymerase I in response to cisplatin induced DNA damage. Mol Biol Cell. 2011 April 1; Vol. 22(7):1070-1079, PMID: 21289084 [4]Cover art: [5]
Cajal body formation correlates with differential coilin phosphorylation in primary and transformed cell lines. J Cell Sci. 2009 Jun 1;122(Pt 11):1872-81. PMID: 19435804 [6]
Relationship of the Cajal Body to the Nucleolus. The Nucleolus. M.O.J. Olson (ed.).
Protein Reviews, 2011, Volume 15, Part 3, 361-380, DOI: 10.1007/978-1-4614-0514-6_16, Springer 2011.
[7]
Fem1b promotes ubiquitylation and suppresses transcriptional activity of Gli1, Biochemical and Biophysical Research Communications, Available online 25 September 2013, ISSN 0006-291X, [8]
