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<center><font face="lucida fax" style= "color:Yellow" size="+10">'''Qianben Wang Laboratory'''</font>
<center><font face="lucida fax" style= "color:Yellow" size="+10">'''Qianben Wang Laboratory'''</font>
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Revision as of 12:53, 10 November 2011

Qianben Wang Laboratory


Laboratory of Cancer Epigenomics


Department of Molecular and Cellular Biochemistry, The Ohio State University


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A histone modification governs androgen receptor to bind to regulatory regions of a class of cell cycle genes in castration-resistant prostate cancer but not in androgen-dependent prostate cancer. This leads to over-expression of cell cycle genes and accelerated growth of castration resistant prostate cancer.