Min-Ho Kim Lab

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[[Min-Ho Kim Lab | <font face="trebuchet ms" style="color:#ffffff"> '''Home''' </font>]] &nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;
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[[Min-Ho Kim Lab:Lab Members | <font face="trebuchet ms" style="color:#ffffff"> '''Lab Members''' </font>]] &nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;
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<h3>Overview</h3>
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<h3>Welcome to the Min-Ho Kim's research group at Kent State University</h3>
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<font size=3>Welcome to the Min-Ho Kim's research group at Kent State University. The major research interest in our laboratory is to (1) understand biological mechanism by which immune cell trafficking contributes to the pathogenesis of chronic inflammatory diseases, (2) apply micro/nano-engineered biomaterials to engineer inflammatory environmental cues, and (3) develop clinically feasible therapeutics to promote the resolution of non-healing chronic wounds. Our laboratory utilizes and combines interdisciplinary approaches of immuno-biology, stem cell biology, cellular and tissue engineering, and nano-bioengineering.</font>
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<font size=3> The major focus of research interests in our laboratory is to develop therapeutics to promote the resolution of non-healing chronic wounds based on precise understanding of inflammatory response to tissue injury and infection. Many non-healing chronic wounds have been associated with chronic inflammation. A more detailed understanding of mechanisms controlling the inflammatory response and how inflammation directs the outcome of the healing process will provide therapeutics for tissue repair in chronic inflammatory diseases. Our long-term goals are to (1) understand the molecular and cellular mechanisms by which local inflammatory environmental cues alter phenotypic switch of tissue infiltrating innate immune cells and these cells functionally interact with mesenchymal stem cells, and (2) develop selective strategy to engineer microenvironmental cues towards tissue regeneration by utilizing micro/nano-engineered biomaterials that enable spatio-temporal control of inflammatory response. To achieve this goal, we combine interdisciplinary approaches of immuno-biology, stem cell biology, cellular and tissue engineering, and nano-bioengineering. </font>
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* Dr. Kim is selected as a recipient of Farris Family Innovation Award to engage research on developing nanothermotherapy platform to target bacterial biofilms in diabetic wounds (Apr. 2013)
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* Dr. Kim is selected as a recipient of Kent State Postdoc Seed Program Award for collaborative projects with Dr. Chris Malcuit (Kent State) and Dr. Fayez Safadi (NEOMED) (Nov. 2012)
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<h3>Positions Available</h3>
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Graduate research assistant positions are available. Highly motivated students are encouraged to send their CVs to Dr. Min-Ho Kim via email at mkim15@kent.edu.
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Home        Lab Members        Publications        Research        Teaching        Contact        News       


Welcome to the Min-Ho Kim's research group at Kent State University

The major focus of research interests in our laboratory is to develop therapeutics to promote the resolution of non-healing chronic wounds based on precise understanding of inflammatory response to tissue injury and infection. Many non-healing chronic wounds have been associated with chronic inflammation. A more detailed understanding of mechanisms controlling the inflammatory response and how inflammation directs the outcome of the healing process will provide therapeutics for tissue repair in chronic inflammatory diseases. Our long-term goals are to (1) understand the molecular and cellular mechanisms by which local inflammatory environmental cues alter phenotypic switch of tissue infiltrating innate immune cells and these cells functionally interact with mesenchymal stem cells, and (2) develop selective strategy to engineer microenvironmental cues towards tissue regeneration by utilizing micro/nano-engineered biomaterials that enable spatio-temporal control of inflammatory response. To achieve this goal, we combine interdisciplinary approaches of immuno-biology, stem cell biology, cellular and tissue engineering, and nano-bioengineering.

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